1	EMP 192 Exam 2 Review
2	Increasing Disease Risk Genetics Cannot be controlled, will affect whoever has those genetics, regardless age Environmental Lifestyle Age Gender
3	Cell Structure Cell Membrane Cytoplasm Nucleus Mitochondria BE ABLE TO LIST ALL
4	Immunologic Disorder Rheumatic Fever Asthma Allergies
5	Age How Does the disease process change as we get older
6	Family Links Disease tends to cluster in families primarily due to the fact that they all have the same genetic predisposition. Environment and Lifestyle will play a factor, but can be controlled.
7	Dynamic Blood Pressure Preload – amount of blood returning to the heart Can be helped by IV bolus of a crystalloid solution. Cardiac Contractile Force – rubber band theory Increased response with catecholamine's Afterload – the pressure in which the heart must overcome upon contraction Determined by peripheral vascular resistance
8	Be Aware of Shock Know primary problem, presentation, and corrections to the main shock types. Cardiogenic Hypovolemic Neurogenic Anaphylactic Septic
9	The Body’s Response How does the body respond to volume depletion that is caused by Hypovolemic shock?
10	M O D S Primary MODS Injury to organ tissue is due to inadequate perfusion. Secondary MODS Organ death occurs from an exaggerated response from those primed cells. PLEASE UNDER STAND MULTIPLE ORGAN DYSFUNCTION SYNDROME.
11	Compensation How will the body compensate for a hypovolemic state. KNOW ALL FACTORS OF COMPENSATION. (Vol. 1 page 212) Angiotensin, ACE, and Renin might be important.
12	Since we are talking SHOCK Compensated Decompensated Irreversible
13	Back to MODS Why is interleukin 1 important in MODS. You have learned it. Can be found in MODS section as well as part 3 in the immune/inflammatory areas.
14	Compensated Shock How will the body be able to compensate?
15	Metabolism Anaerobic phase Aerobic phase How are these different, and what happens in each???
16	Nasty Bacteria Must attach to host cells Release exotoxins during growth Examples include-botulism and tetanus Release endotoxins upon death What do they consist of??? What is the term for the systemic spread of toxins??
17	Viruses Capsid - ????? They consis of strands of - ??? The do not attach to the host cell like bacteria, instead they invade and live inside the host cell. Why is this BAD???
18	Infectious Agents Bacteria and Viruses are common here. What are the 3 others. One is similar to a virus. WHY??
19	Three Lines of Defense Anatomical Barriers Sebaceous Glands secrete fatty and lactic acid Sweat, Tears and Saliva contain bacteria attacking enzymes. Inflammatory Immune
20	Three line of Defense Anatomical External, Non-specific Inflammatory Internal, Non-specific Immune Internal, Specific Why are each classified as internal vs. external and specific vs. non-specific. Think about location and action.